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Also, minocycline partially inhibits caspase-3 activation and photoreceptor degeneration after photic injury.

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Finally, we showed that minocycline promoted neuroprotective phenotype alternative activation of microglia macrophages, which is involved in the neurovascular remodeling after stroke.

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Early minocycline treatment against reperfusion injury significantly promotes neurovascular remodeling during stroke recovery by reducing brain tissue loss, enhancing tjp expression in ischemic brains and facilitating neuroprotective phenotype alternative activation of microglia macrophages.

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minocycline, the active substance of the drug, is a semi synthetic antibiotic from the group of tetracyclines.

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